7 edition of The neuropathology of temporal lobe epilepsy found in the catalog.
|Series||Maudsley monographs ;, no. 31|
|LC Classifications||RC372.5 .B78 1988|
|The Physical Object|
|LC Control Number||87031339|
It is clinically associated with the syndrome of mesial temporal‐lobe epilepsy and is frequently associated with drug resistance. Granule cell dispersion is commonly observed in the context of epilepsy‐associated hippocampal : Maria Thom. Anterior temporal lobectomy for mesial temporal sclerosis is a very effective measure to control seizures, and the probability of being seizure-free is approximately %. However, 30% of patients still experience seizures after surgery. In neurosurgery there are several situations that require transgression of the temporal cortex. For example, a subset of patients with temporal lobe.
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Pathologic findings in surgical resections from patients with temporal lobe epilepsy include a wide range of diagnostic possibilities that can be categorized into different groups on the basis of etiology.
This paper outlines the various pathologic entities described in temporal lobe epilepsy, including some newly recognized epilepsy-associated tumors, and briefly touch on the recent Cited by: The title is misleading in the sense that more than simply the neuropathology of epilepsy is covered—the book represents a truly multidisciplinary approach.
In general, it is clear that each chapter is based on the extensive personal experience of its respective authors; the quality of the writing is high and the explanations are : J. Nicoll. temporal lobe epilepsy, including some newly r ecognized epilepsy-a ssociated tumors, and brieﬂy touch on the recent classiﬁcation of focal cortical dysplasia.
Neuropathology of temporal lobe epilepsy. Oxford ; New York: Oxford University Press, (OCoLC) Online version: Bruton, C.J. Neuropathology of temporal lobe epilepsy. Oxford ; New York: Oxford University Press, (OCoLC) Document Type: Book: All Authors / Contributors: C J Bruton.
the clinical syndrome of mesial temporal lobe epilepsy (MTLE) but can be seen in other epilepsy syndromes as well as in ageing without epilepsy The neuropathological features have been recognised for over a century (for historical review see Thom ) but its cause, and in turn. This book provides the first in-depth examination of the Maudsley Hospital program.
In addition to investigating the results of the operations, it presents a new classification of the pathological abnormalities found in temporal lobe surgery, and gives an unbiased assessment of the technique's successes and failures, along with a guide to the Author: C.
Bruton. Introduction. Temporal lobe epilepsy (TLE) is the most common form of focal epilepsy .In medical centers where epilepsy surgery is performed, neuropathologists often encounter temporal lobe Cited by: Temporal lobe epilepsy (also called mesial temporal lobe epilepsy), the most frequent form of partial epilepsy in adults, is clinically characterized by alterations in consciousness combined by.
Increasing attention is being paid to the relationship of cerebral lesions to the epilepsies. Gastaut and Fischer-Williams 27 consider that cases of so-called functional or idiopathic epilepsy (which represent a fault in cerebral function rather than in structure) constitute only about 5% of cases and that most cases of epilepsy have underlying cerebral by: Temporal lobe epilepsy (TLE) is the most common form of focal (partial) epilepsy.
It can be hard for people with TLE to become completely seizure free with seizure medicines alone, though medicines may lower the number of seizures.
People with drug-resistant medial temporal lobe epilepsy have a higher risk for memory and mood difficulties. Temporal lobe epilepsy (TLE) is a chronic disorder of the nervous system characterized by recurrent, unprovoked focal seizures that originate in the temporal lobe of the brain and last about one or two minutes.
TLE is the most common form of epilepsy with focal seizures. A focal seizure in the temporal lobe may spread to other areas in the brain when it may become a focal to bilateral lty: Psychiatry, Neurology. For children Lee: the Rabbit with Epilepsy by Deborah Moss. Part of "The Special Needs Collection" for ages Published24 pages.
Explains epilepsy in a reassuring way for newly diagnosed children, their siblings and friends. Special People, Special Ways by Arlene Maguire. Published32 pages. A colorfully illustrated book about children with disabilities for.
The temporal lobes are the most common brain region to develop epileptogenicity. Historically, "uncinate fits" were first described by Hughlings Jackson in the 19th Century linking seizures presenting as "dreamy states" to lesions in the uncus of the temporal lobe.
In the midth century, the term "psychomotor epilepsy" was introduced by Gibbs and Gibbs to describe the characteristic. Even earlier, Corsellis had written his review chapter on neuropathology of temporal lobe epilepsy, published in Modern Trends in Neurology (Corsellis, ).
This contained a full description, including microphotographs, of what is today known as focal cortical dysplasia; again the reminiscence of tuberous sclerosis was by: The neuropathology of symptomatic and idiopathic epilepsy is presented.
New methods of investigation have disclosed that more cases of epilepsy are symptomatic — even hippocampal sclerosis is now detectable in vivo by MR-scan.
It still remains to be defined whether hippocampal sclerosis is the cause or the effect of by: 1. The EEG localizes the epileptogenic focus in the medial portion of the temporal lobe. Because TLE is refractory to drugs, it is often treated by resection of the temporal lobe including the hippocampus and surrounding area and the amygdala.
Examination of temporal lobectomy specimens reveals pathology in. Surviving Wonderland: Living with Temporal Lobe Epilepsy by Sharon R. Powell was the most cathartic book I've read in some time.
I learnt things about my own epilepsy, for I have the same type Sharon has. This book is informative to the patient with epilepsy and their caregivers. I highly recommend it/5(15). Temporal lobe epilepsy and Dostoyevsky seizures: Neuropathology and Spirituality Dr Alasdair Coles Religious Seizures The fullest description of what have been termed ‘religious seizures’, comes from the writing of Dostoyevsky (hence the sobriquet ‘Dostoyevsky seizures’), particularly in the.
DA intoxication causes neurological symptoms, including status epilepticus. Many surviving sea lions develop temporal lobe epilepsy. The neuropathology of epileptic sea lions closely matches that of human patients with temporal lobe epilepsy.
DA-intoxicated sea lions are cared for by wildlife rescue centers, but more effective treatments are. Thus Neuropathology of Focal Epilepsies: An Atlas may nicely fill the niche. The book is divided into 15 chapters.
The first five chapters are short, each covering introduction, epidemiology, historical overview of epilepsy surgery, methods of specimen processing, and.
The book will include chapters by physicians, physiologists, radiologists, neurosurgeons, all with special interest in the field of epilepsy.
The description of the pathological features will include immunohistochemical data as well as results of morphometric analysis. Contents: Epilepsy: Epidemiology, Presentation and Classification (D R Fish). Temporal-lobe epilepsy (TLE) is a well-known form of focal epilepsy in adults (Hauser et al.,Berg et al., ).
HS is the most common neuropathologic finding in patients with drug-resistant TLE (Thom,Wieser and ILAE Commission on Neurosurgery of Epilepsy,Blumcke and Spreafico, ).Cited by: The first part of this work concentrates on the concept of mesial temporal lobe epilepsy and its pathophysiology.
The second part addresses the clinical characteristics, in particular the natural evolution of the illness, the different aetiologies and the diagnostic relevance of the ictal semiology. 8th International Summer School for Neuropathology and Epilepsy Surgery (INES ) Print Share.
Print Share. 8th International Summer School for Neuropathology and Epilepsy Surgery (INES ) University Hospital in Erlangen, Germany 26 - 29 July with particular emphasis on mesial temporal lobe epilepsy with hippocampal sclerosis.
Temporal lobe epilepsy represents the largest group of patients with treatment resistant/medically intractable epilepsy undergoing epilepsy surgery. The underpinnings of common forms of TLE in many instances begin in early life with the occurrence of an initial precipitating event.
The first epileptic seizure often occurs after a variable latency period following this by: epilepsy. Subsequently it has become clear that some adults who have another cause for temporal lobe seizures, such as a contusion, acquire this as a second lesion.
The epileptogenic nature of this abnormality is unproven, but it can clearly be a consequence of poorly-controlled temporal Size: 2MB.
Abstract. Generalized convulsive status epilepticus is a life-threatening condition with high mortality; it also causes cellular damage. Systemic physiologic changes in blood pressure, oxygenation, and temperature can cause widespread neuronal injury, but even when systemic physiologic parameters are controlled, status epilepticus can cause focal cell loss in the : Nathan B.
Fountain, Suchitra Joshi. Methods: In 12 epilepsy surgery cases that involved the temporal lobe, we correlated the IrTM, electrocorticography, and neuropathology results. Results: Irritative zones (IZ) had a lower temperature in comparison to the surrounding cortex with normal electric activity (difference in temperature (ΔT) from tomean °C standard.
Epilepsy is the most common serious neurological condition, affecting children and adults, and can occur in a variety of medical settings. It has many causes and many forms, and a variable prognosis. Mortality and morbidity are high, social and legal consequences can stretch well beyond the purely medical, and its management is often poor.
Part of the Oxford Textbooks in Clinical Neurology. Karl‐Heinz Stauder, of Munich, who was vice‐president on the ILAE Executive Committee from towas one of the first to demonstrate a syndrome‐specific correlation between temporal lobe epilepsy and hippocampal sclerosis (Stauder, ).
But he stopped short of identifying hippocampal damage as a cause of by: This chapter is devoted to defining some of the mechanisms that determine the behaviour of cellular membranes, both of neurons and glial cells, during the transition towards epileptic discharges and henceforth during seizures.
We take the view that epileptic events are not the sole result of concerted populations of neurons, but equally important, of the dialogue between neurons, glial cells.
arising from the temporal lobe, and if dysphasia occurs this is a useful lateralising sign indicating seizure origin in the speech-dominant temporal lobe Post-ictal nose-rubbing is commonly seen in temporal lobe epilepsy, and in 90% of cases is ipsilateral to the focus Amnesia is.
Palmer is the Director of Neuropathology at ARUP as well as a Professor of Pathology and the Associate Program Director of the Pathology Residency Program Jump to main navigation Jump to. Neuropathology of Human Epilepsy Abstract.
In this chapter, we briefly review salient features of the pathology of human focal epilepsies. We present human histopathological data and classification of hippocampal sclerosis (HS) as well as findings in long-term epilepsy-associated tumors (LEATs) and focal cortical dysplasias (FCDs).
FAMILIAL MESIAL TEMPORAL LOBE EPILEPSY. Inwe described familial mesial temporal lobe epilepsy (FMTLE) as a benign syndrome with onset in the second to fifth decades of life, no temporal lobe or hippocampal abnormalities on MRI, and no association with febrile seizures (table 1).
16, 17 The nature of the aura, with prominent psychic and autonomic features, suggested mesial temporal Cited by: epilepsy [ep´ĭ-lep″se] paroxysmal transient disturbances of nervous system function resulting from abnormal electrical activity of the brain.
Epilepsy is not one specific disease, but rather a group of symptoms that are manifestations of any of a number of conditions involving overstimulation of nerve cells of the brain. The estimated incidence is 0.
As a prominent member of the British Neuropathological Society, his contribution is remembered in a biennial memorial lecture. His most significant work was on the anatomical aspects of frontal leucotomy (prefrontal lobotomy), and the nature of the structural abnormalities in the. Temporal lobe or psychomotor seizures are caused by abnormal electrical activity in one area of the brain.
Learn about the symptoms. Book reviewed in this article: Ulrich J. (Ed.) Histology and Histopathology of the Aging Brain S. Karger Imura H. (Ed.) Neuroendocrine Control of the Hypothalamo–Pituitary System S. Karger Bruton C. The Neuropathology of Temporal Lobe Epilepsy.
Maudsley Monographs No Functional Neuropathology of Temporal Lobe Epilepsy / Cheolsu Shin --Ch. Visual Analysis in Mesial Temporal Sclerosis / Graeme D. Jackson -- Ch. Mesial Temporal Sclerosis: Magnetic Resonance-Based Hippocampal Volume Measurements / Clifford R.
Jack, Jr. -- Ch. " Temporal lobe epilepsy is a form of focal epilepsy with seizures originating from the brain's temporal lobe. Statistics method shows networks differ in epileptic brains Chayasirisobhon, "The mechanisms of medically refractory temporal lobe epilepsy," Acta Neurologica Taiwanica, vol.
Temporal-lobe epilepsy: Epilepsy that is characterized by abnormal electrical activity in the temporal lobe of the brain. This activity does not cause grand mal seizures; rather, it causes unusual behaviors and patterns of al lobe epilepsy may, for example, cause sudden outbursts of unexpected aggression or agitation, or it may be characterized by aura-like phenomena.Temporal lobe epilepsy, as discussed in this chapter, is a focal epilepsy that involves primarily the limbic structures of the medial temporal lobe (amygdala, hippocampus, and entorhinal cortex).
In recent years animal models have been developed that mirror the pathology and pathophysiology of this disease. This chapter reviews the human condition, the structural and physiological changes that.